Garrido Pérez, NuriaResearcher from the University of Zaragoza

Nuria Garrido Pérez

Centre: Dpto. Bioquimica y Biologia Molecular y Celular
Institution: University of Zaragoza, Zaragoza (Spain)
Position: Researcher from the University of Zaragoza
E-mail: ngarrido@unizar.es
Phone: 976 762843
Profile Click here

¿Quién soy?
I completed my doctoral thesis in the Department of Biochemistry and Molecular Biology at the University of Zaragoza on the mechanisms regulating the expression of mammalian mitochondrial DNA, under the supervision of Julio Montoya Villarroya and J. Antonio Enríquez Domínguez. Afterwards, I moved to the Institute of Medical Technology in Tampere, Finland, where I worked on mechanisms of mitochondrial DNA maintenance. In 2004, I returned to Spain with a contract as Assistant Professor and rejoined the Genoxphos group in the Department of Biochemistry and Molecular Biology and Cellular Biology, where I continued working on mechanisms regulating OXPHOS function. Currently, I am a Contracted Professor and part of the Mitochondrial Biogenesis and Pathology group at the University of Zaragoza.

Mi perfil investigador
As I have mentioned, I am currently a Contracted Professor in the Department of Biochemistry and Molecular Biology and Cellular Biology at the University of Zaragoza. I study the role of mitochondrial biogenesis in diseases such as Alzheimer’s, Parkinson’s, and, at this moment, Down syndrome. My interest lies in investigating the role of mitochondria, the OXPHOS system, and mitochondrial DNA in these pathologies, while also gaining a better understanding of the mechanisms leading to their development. I am also very interested in exploring potential therapies aimed at improving mitochondrial functionality for these diseases.

Why my research is important
My research line is important because the role of mitochondria in pathologies such as Alzheimer’s, Parkinson’s, and Down syndrome is still not well understood. We do not know whether the OXPHOS dysfunction observed in these patients is a cause or a consequence of these diseases. Above all, our goal is to propose potential therapies that improve mitochondrial biogenesis and functionality, thereby enhancing the prognosis and quality of life for these patients.

Know more about me and my research
–  https://bifi.es/es/bioquimica-bmc/

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